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44 pages 1 hour read

Jason Fung

The Obesity Code: Unlocking the Secrets of Weight Loss

Nonfiction | Book | Adult | Published in 2016

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Part 1Chapter Summaries & Analyses

Part 1: “The Epidemic”

Part 1, Chapter 1 Summary and Analysis: “How Obesity Became an Epidemic”

Chapter 1 traces the origins and evolution of the obesity epidemic. Combating obesity requires understanding its root causes. Conventional wisdom holds that consuming too many calories and exercising too little leads to weight gain. This belief gave rise to the maxim “Eat Less, Move More. Fung problematizes this approach to obesity by distinguishing between the proximate and ultimate causes of weight gain. Eating too much may be immediately responsible for weight gain (the proximal cause), but it is not what starts the chain of events (the ultimate cause). Moreover, obesity is not a personal failing or choice, as the maxim implies. Women have almost 50% more body fat than men, even though men generally consume more calories (12). Women are not lazier than men, nor do they lack self-control. Rather, hormones make it more difficult for women to burn calories. Despite the clear problems with the “Eat Less, Move More” strategy, government organizations continue to promote it as the gold standard for weight loss.

Starting in the late 18th century, health experts posited a link between weight gain and carbohydrates (sugar molecules found in sugary and starchy foods). The increased prevalence of coronary disease in the mid-20th-century (largely attributable to longer lifespans) led to the replacement of dietary fats with other food types, namely, carbohydrates. However, carbohydrates could not both be healthy (low in fat) and unhealthy (fattening). Thus, a new narrative emerged, presenting calories, rather than specific foods (except fat), as fattening. Despite the lack of scientific evidence, US dietary guidelines from 1977 and 1980 promoted this narrative by promoting the consumption of breads, potatoes, and pastas with their widely distributed food pyramid. The American Heart Association (AHA) disseminated the same message, which soon spread globally. Statistics show that Americans followed their doctors’ advice, consuming more refined carbohydrates, while cutting back on meat, eggs, and other fats. Sugar consumption increased as the health conscious focused on low-fat and low-cholesterol foods. Fung posits a connection between these new dietary guidelines and rising obesity rates.

Part 1, Chapter 2 Summary and Analysis: “Inheriting Obesity”

Chapter 2 probes the nature versus nurture obesity debate. Obesity runs in families. Children who are overweight often have overweight siblings. Moreover, obese children are 200 to 400% more likely to become obese adults than their non-obese peers (21). These statistics have led some scientists to believe that obesity is inherited. Other scientists have questioned this assumption, positing that environment, and not genetics, plays a key role in weight gain. Families tend to eat the same foods and have similar attitudes about when and how much to eat. In short, they share an obesogenic environment. Calorie-based theories point to varied environmental factors to explain the obesity epidemic, including diet, eating more, computer time, and the increased consumption of fast foods, sugar, and high-fructose corn syrup.

Citing a 1986 Danish study of 540 adult adoptees, Fung complicates the genetics versus environment debate. The study found no relationship between the weight of the adoptees and that of their adoptive parents, suggesting that environment plays little role in determining body weight. By contrast, there was a strong correlation between the weight of the adoptees and their biological parents. A 1991 study of fraternal twins raised in different environments found a 70% familial variance for obesity (24). As Fung observes, however, obesity rose dramatically within a single generation. Since genes cannot change in this short period, they cannot be solely responsible for the obesity epidemic. He argues instead that obesity results from a combination of genetic and environmental factors. Hormonal imbalances and high blood insulin levels account for 70% of the tendency to gain weight, leaving 30% open to environmental control.

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